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These alterations contribute to mitochondrial dysfunction, insulin resistance (IR), and the pathogenesis of sarcopenia [39, 81]

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Treatment sessions are typically less than an hour

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Together, these data suggest that GLP-1R agonism and GIPR antagonism may exert their effects on energy balance by downregulating signalling from DVC C35 GABA4 and C35 Glut10 neurons to hypothalamic feeding circuits