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GLP-1-based therapies for diabetes, obesity and beyond

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Why are therapeutic injection claims frequently denied

Within the pancreas, GIP and GLP-1 together promote -cell proliferation and inhibit cell death, thereby resulting in pancreatic -cell mass-increase, while GIP enhances postprandial glucagon response and GLP-1 suppresses it [9, 10]

Our laboratory experiments showed that the pomegranate extract can reduce the production of TMA

For patients with severe obesity or obesity-associated comorbidities, this difference can be clinically meaningful, reinforcing the preference for injectable therapies in high-risk patient populations