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The exact mechanism of action in a tendinopathic condition such as medial epicondylitis is poorly understood, as the effects of corticosteroid are predominantly anti-inflammatory

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Disruption of proteostasis is now recognized as a core pathological mechanism across cardiomyopathies, ischemic heart disease, arrhythmias, and heart failure, linking molecular protein quality control failure to tissue remodeling and clinical disease progression

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