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These effects have positioned GHK-Cu as a key subject of investigation in wound-repair models, inflammation-related signaling studies, and extracellular matrix regulation research

any bridging therapy must be individualized within the boundaries of periconception contraindications (170)

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Key pathways, including Wnt/-catenin, bone morphogenetic protein 2/Smad, phosphoinositide 3-kinase/Akt and protein kinase A, play a role in this

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