In liver, increased hepatic DAG levels induced the translocation of PKC (the primary nPKC isoform in liver) to the plasma membrane and inhibited IRTK tyrosine kinase activity by phosphorylating it at Thr1160, which reduced insulin-stimulated phosphorylations (activations) of IRS2, PI3K, and Akt2 [108,109]
103,104 In the diagnostic practice of AP, several key considerations warrant particular attention
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