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The article says GLP-1 therapy may slightly lower resting metabolic rate in some individuals as a compensatory mechanism during weight loss

Studies have shown that in a sepsis mouse model, inhibiting Hmox1 effectively activates the FSP1/CoQ10/NADPH pathway, significantly increasing autophagy activity and suppressing ferroptosis in alveolar epithelial cells, thereby alleviating lung injury in mice (Li et al., 2025b)

The active ingredient is the same semaglutide molecule, but a compounded preparation is not an FDA-approved finished drug product

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The authors then treated dorsal root ganglion neurons with exendin-4