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For individuals with Wilson's disease (a genetic disorder causing copper accumulation) or other copper metabolism disorders, theoretical caution is warranted, though even in these populations topical GHK-Cu is unlikely to pose significant risk given the minimal systemic absorption

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CSAP, chronic stable angina pectoris

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Plasma levels of GHK have been documented to decline from approximately 200 ng/mL at age 20 to roughly 80 ng/mL at age 60, a 60% drop [1]

if you're experiencing inflammation, look for causes beyond the GHK-CU molecule