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Trees for Life Journal

Effects of growth hormone treatment on the leptin system: interactions with body composition and energy expenditure

Once the combined oxidative capacity of this system is exceeded, LD expansion becomes insufficiently protective and may convert into a feed-forward mechanism sustaining NEFA accumulation, ROS generation, bioenergetic collapse, and necroinflammation ( 3.6 Mitochondria and secretory granules Because pancreatic injury is ultimately expressed through disturbed secretion and premature enzyme activation, the consequences of mitochondrial dysfunction must also be considered within the apical secretory domain

Moreover, GSTM2 overexpression reversed methionine choline-deficient diet (MCDD)-induced steatosis