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Meanwhile, TMAO can significantly upregulate the expression level of PIEZO1 and enhance the sensitivity of chondrocytes to mechanical loading, and this effect may be closely related to the pathogenesis of OA (Zhuang et al., 2023)

45,74 In summary, expanding practical guidance for the management of GI adverse effects is critical to ensuring treatment continuity and maximizing clinical benefit

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A variety of cellular stresses, including DNA damage can cause the induction of cellular senescence, which represents a multifaceted and highly heterogeneous cellular state characterized by irreversible growth arrest and importantly the secretion of a multitude of factors, including proteases and chemokines 2