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Ho JY, Kraemer WJ, Volek JS, Fragala MS, Thomas GA, Dunn-Lewis C, Coday M, Hakkinen K, Maresh CM

The present review provides (1) an overview in the pathophysiology of NAFLD, including the potential involvement of GLP-1 and glucagon, (2) an introduction to the currently available GLP-1RAs and (3) outlines the potential of emerging GLP-1RAs and GLP-1/glucagon receptor co-agonists in the treatment of NAFLD

[1] Some common antioxidants include vitamin C, E, beta-carotene, selenium, and polyphenols

Transcriptional regulation by Nrf2

On the other hand, increasing glucose oxidation reduces lactate and H+ ion accumulation, thereby preventing intracellular acidosis in myocardial cells during ischemia, which aids in the faster recovery of ischemic heart function during reperfusion

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