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These antibodies contribute to glandular injury via complement-dependent and antibody-dependent cell-mediated cytotoxicity, and promote the release of cytokines such as IL-6, IL-1, and IL-8, which enhance lymphocyte accumulation and the local inflammatory response

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Participants with hepatic impairment must have been on a stable dose of medication or treatment regimen (e.g., no new medications and no changes to current medications within 14 days of screening) and were excluded if they had clinical signs of acute hepatitis, records of biliary obstruction or other causes of hepatic impairment not related to parenchymal disorders or diseases, a history of hepatic encephalopathy Grade 3 or presence of hepatic encephalopathy Grade 2, gastroesophageal varices not managed according to standard of care for patients with cirrhosis or with bleeding within 90 days prior to screening, acute or exacerbation of chronic liver failure or history of liver transplantation

CJC-1295 with DAC is the longer-acting form

Scar tissue was assessed using a delayed enhancement inversion-recovery sequence (slice thickness 8 mm, echo time 1.4 ms, field of view 300360 mm, no slice gap

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