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however, since this work was done in vitro , it is most unlikely that the sDPP4-induced impairment of insulin action is due to an increased bioavailability of any DPP4 substrate

This "fatty bubble" acts as a protective delivery vehicle

Here is what the data shows

10.1038/s41598-018-27221-7 78 KirmitA.KaderS.AksoyM.BalC.NuralC.AslanO

Our study preliminarily predicted that SIRT1 may be a core target of GHK in the treatment of UC by utilizing network pharmacology and molecular docking