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Secondary outcomes included nonfatal MI, nonfatal stroke, HHF and renal composite outcome, defined in most trials as a composite of progression to end-stage renal disease or a sustained decrease of at least 40% in estimated glomerular filtration rate (eGFR)

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Literature on BPC-157 research includes model-specific work involving tendon fibroblast behavior, angiogenesis, nitric oxide signaling, endothelial context, and FAK-paxillin pathway discussion [4] [6] [9]

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Direct inhibition of IRF-dependent transcriptional regulatory mechanisms associated with disease