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Quantitative proteomic study of the plasma reveals acute phase response and LXR/RXR and FXR/RXR activation in the chronic unpredictable mild stress mouse model of depression

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Preclinical models suggest that GHK-Cu may modulate the expression of TGF-, VEGF, FGF, and IGF-1, contributing, in laboratory settings, to fibroblast activity, angiogenesis, and extracellular matrix remodeling [1]

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The cell culture experiments in this study indicate that NFT concentrations in the range of 550 M were optimal for the induction of de novo glutathione synthesis