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Mechanistically, ROS exert their pro-tumorigenic effects mainly through activation of ERK/MAPK, PI3K/AKT, and STAT3 pathways representing a paradigm shift from viewing ROS solely as cytotoxic molecules to recognizing them as key signaling intermediates in HCC development

S MA et al

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The inflammaging process involving the accumulation of pro-inflammatory senescent cells triggers immunosuppressive mechanisms to maintain tissue homeostasis [122]

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