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Excess ROS not only damages tissues but also serves as a second messenger for immune activation for instance, ROS can activate NF-B and stabilize HIF-1 in immune cells, skewing macrophages to a pro-inflammatory phenotype (42)

Multinuclear magnetic resonance spectroscopy for in vivo assessment of mitochondrial dysfunction in Parkinson's disease

In summary, these experiments demonstrated that (i) GSH can replace the natural bound factor, (ii) that Cys-Gly (CG), the initial breakdown product of GSH 9 , also binds at the I site and (iii) that GSH binds more strongly than CG (Fig