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After infectious or sterile stimuli, SLC15A4 in DCs is recruited to phagosomes, SLC15A4deficient DCs infected with Salmonella typhimurium show reduced caspase-1 cleavage and IL-1 production, this correlates with peripheral NLRC4 inflammasome assembly and increased autophagy

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The presence of cholesterol in the niosomes formulation was considered important for keeping the assemblage integrity and physical stability and rigidity of these vesicles 60,61,62

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