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Furthermore, in mice that were fed a short-term high fat (45%) diet, etomoxir stimulated glucose oxidation, peripheral glucose disposal, and led to elevated circulating fatty acids and circulating triglycerides (TGs) within 5 h of treatment and after several days hepatic steatosis was induced [60]

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In contrast, the pathophysiology associated with the decreases in NM amounts in SN and LC remains not well understood (Hirsch et al

What makes this combination distinct from single-peptide studies