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Because rapamycin, which extends mouse lifespan 31 , can reverse age-related diastolic dysfunction and restore a more youthful metabolome in the aged myocardium 32 , we next evaluated metabolomic changes in Young, Old Con, and Old mAb female-treated hearts

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Individuals with type 2 diabetes treated with GLP-1s have demonstrated improvements in inflammatory signaling and mitochondrial redox balance, including reduced reactive oxygen species production, improved mitochondrial membrane potential, and increased oxygen consumption

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