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Targeting Aggregation of Wilde-Type p53 and Mutant p53 with ReACp53 As a Novel Therapeutic Concept for AML

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By binding to actin monomers, it creates a reserve of cellular building blocks that are rapidly deployed to help cells move into a damaged area to begin reconstruction

doi: 10.1016/j.jdds.2017.06.001 78 GopalKVRaoGRKumarYH