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However, their precise mechanisms of action remain incompletely understood

The presence of oxidized phospholipids and lysophospholipids, together with pro-inflammatory proteins, in dysfunctional HDLs, obstacles cholesterol efflux, resulting in LDL oxidation

Recently, PCBPs were shown to maintain nonheme iron cofactors in cytosolic enzymes, especially when iron becomes scarce [37]

43 Another study, which compared Caucasian and Chinese populations indicated the increased susceptibility of the former to APAP hepatotoxicity reflects the lower CYP 2E1 and 2D6 activity in the latter