Neuropsychopharmacology 39 (13), 30093017
Lopez-Siles et al., 2016
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Far from a mere byproduct, TMAO can actively accelerate CKD progression by fueling renal inflammation and fibrosis, potentially creating a vicious cycle of TMAO accumulation and ongoing kidney damage [4,5]
doi: 10.1016/j.ejphar.2023.175875 Summary Keywords ferroptosis, Parkinsons disease, neuronal death, oxidative stress, lipid peroxidation, epigenetics Citation Tu R, Han Z, Zhang H, Jia X, Sun T, Liu H, Li J, Tang M and Wang S (2025) From pathogenesis to treatment: the emerging role of ferroptosis in Parkinsons disease
GAD levels and muscimol binding in rat inferior colliculus following acoustic trauma