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SU11274, another ATP-competitive c-MET selective inhibitor, blocked the constitutive phosphorylation of c-MET and downstream kinases and induced G2/M cell cycle arrest and significantly suppressed cell growth in a dose-dependent manner [42]

The separation and overlap of IGF-I and insulin functions are, in part, due to the ability for both ligands to bind to these highly homologous receptors with different affinities

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Continuous use without breaks may reduce its long-term effectiveness and increase adaptation