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In the hippocampal CA1 region, VPA-exposed rats showed significant SV depletion compared to controls ( P 0.05 )

Mito-TEMPO, a mitochondrion-targeted antioxidant, significantly rescued doxorubicin (DOX)-induced myocardial ferroptosis, substantiated that mitochondrial oxidative damage underlies ferroptosis-induced cardiac injury [57]

Such validation efforts not only strengthen the credibility of predictive models but also lay the theoretical groundwork for their clinical application

Hum Psychopharmacol

New York: Wiley

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