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IGF-1 displays more binding sites for SHP2 (a phosphatase related to growth) and seems more prone to recruit Cbl [121] (an E3 ligase that targets the receptor for ubiquitination and destruction) and thus may explain a different regulatory mechanism not mediated by serine phosphorylation, and thus not so sensitive to metabolic derangements

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While this is an effective method, it hasnt been carefully studied in clinical trials (22)

The peptide has a half-life of approximately 24 hours, but its downstream effects on AMPK phosphorylation persist for 1224 hours