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This heterogeneous nature of HNSCC depends on cell origin on anatomical location and etiological agent

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GHK-Cu enhances wound healing through multiple mechanisms including stimulation of angiogenesis (essential for delivering nutrients and oxygen to healing tissue), enhancement of collagen and glycosaminoglycan synthesis (providing structural matrix for tissue reconstruction), and modulation of inflammatory responses (preventing chronic inflammation while supporting protective immune functions)

doi: 10.1016/j.yfrne.2014.04.004 5 TuressonCOFallonWMCrowsonCSGabrielSEMattesonEL

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