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Conclusions Vitiligo is a complex disease whose pathogenesis results from the interaction of different factors, including genetic predisposition as well as oxidative and psychological stress

Kimberlin, D

Accumulating data suggests that the cellular content of oxidized nicotinamide adenine dinucleotide (NAD + ) content is frequently reduced in target tissues due to enhanced NAD + consumption under metabolically adverse scenarios related to impaired insulin signaling conditions, i.e., diabetes, and obesity [19]

Additionally, further cell experiments indicated that these nanomicelles induced apoptosis in tumor cells through downregulation of Pro-caspase9 and Bcl-2 and upregulation of cleaved-caspase9 and Bax via the mitochondrial pathway

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