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10.1016/j.diabres.2021.108935 Diabetes Res Clin Pract
-catenin blocks its transcriptional complex with TCF/LEF through the interaction with FOXO-induced ROS 90
Instead it works through a distinct set of mechanisms: NFB inhibition: KPV's primary action is blocking NFB, the master switch that drives inflammatory gene expression in immune and epithelial cells
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GH production depends upon the availability of IGF-1
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