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Global genomic profiling reveals an extensive p53-regulated autophagy program contributing to key p53 responses

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A 2023 study revealed that HSV-1 infection enhances the ubiquitination and degradation of NRF2 by the E3 ubiquitin ligase Keap1, disrupting cellular redox homeostasis and promoting ferroptosis in the mouse central nervous system

[1] The disorder results from an antibody, called thyroid-stimulating immunoglobulin (TSI), that has a similar effect to thyroid-stimulating hormone (TSH)