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Effect of s-allylcysteine on oxidant-antioxidant status during n-Methyl-N-Nitro-N-Nitrosoguanidine and saturated sodium chloride-induced gastric carcinogenesis in wistar rats

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In fact, systemic administration of MOTS-c has been shown to reverse diet-induced insulin resistance by enhancing glucose uptake in the skeletal muscle of C57BL/6 mice and to mitigate T cell-mediated insulin deficiency by ameliorating autoimmune pancreatitis in nonobese diabetic (NOD) mice 7,8