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Blocking the tumor endothelial marker TEM8/anthrax toxin receptor 1 using antibodies raised against the extracellular domain inhibited angiogenesis, decreased growth of human tumor xenografts and increased the effect of anticancer drugs [322]

These mechanisms are closely linked to pathological changes, including ion channel dysregulation, aging, hypoxic-stress injury, fibrosis, and autonomic nervous dysfunction (Schmig et al., 1995

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