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The biomarker phosphatase and tension homolog (PTEN) interacts closely with other downstream factors in the RTK pathway responsible for cell cycle progression, growth, and metabolism [32]

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Secondary immunodeficiency was found in 10% of patients with RA It was more common in patients with earlier RA onset, seronegative disease and rituximab exposure Whole-exome sequencing identified 17 putatively pathogenic rare variants in inborn errors of immunity (IEI)-associated genes in 15 of 70 patients 41.2% of variants affected genes involved in canonical NF-B signalling Two patients harboured variants previously reported as pathogenic The findings suggest that, in a subset of patients, immunodeficiency may not be explained by immunomodulatory therapy alone

Ferroptotic pores induce Ca2+ fluxes and ESCRT-III activation to modulate cell death kinetics