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However, another study by Cantini et al., used GLP-1(7-36), GLP-1(9-36) and Liraglutide to induce apoptosis in unstressed hASC, as assessed in annexin-V expression on the surface, though only the effect of Liraglutide and GLP-1(7-36) was partially reversed by the GLP-1-R-Antagonist Exendin(9-36), supporting receptor-specific activity [12]

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The LR3 in the name is what sets this variant apart