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In Alzheimers disease research, GHK-Cu has been studied for its capacity to reduce A aggregation (measured by ThioT fluorescence and atomic force microscopy) through copper chelation from amyloid-promoting Cu-A complexes, and to reduce tau hyperphosphorylation markers

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The concentration of APs was markedly increased in Fru (+ 86%) and Fru + amlodipine (+ 20%) in comparison with BSA (Fig

10.1016/j.expneurol.2015.02.032 Exp

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