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These findings indicate that mammalian hepatocytes, unlike microbes, must have a constitutive alternative mechanism of supporting RNR activity and redox homeostasis that can sustain hepatocyte physiology in the complete combined absence of both TrxR1 and Gsr

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Since then, the knowledge of redox biology has undergone significant advancements and the concept has been redefined to account for the broad implications of redox homeostasis [1,3,4,5]

Oral supplements, intravenous treatments, and combined therapy protocols should always be guided by qualified medical professionals