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In this paper, we evaluated the hepatoprotective effects of SAG in a CCl4-model of liver injury, in which, the liver microsomal oxidizing systems related to cytochrome P-450 produce reactive metabolites of CCl4, including trichloroperoxyl radicals (CCl3O3) or trichloromethyl radicals (CCl3)

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To enhance statistical power of the study, researchers employed the intention-to-treat (ITT) method for data analysis