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Sildenafil acts on adipocytes, possibly through cGMP-dependent protein kinase I and mechanistic/mammalian target of rapamycin (mTOR) signaling pathways, browning subcutaneous white fat, thus increasing energy expenditure (232)

No elevated rates in trials

The decision to continue, modify, or discontinue semaglutide should be individualized, weighing the cardiovascular and metabolic benefits of the medication against the impact of gastrointestinal side effects on patient well-being and treatment adherence

Electrolytes support nerve signaling and muscle function, both of which decline when fluids are low

A: GLP-1 therapy reduces appetite and slows digestion, leading to reduced food intake and weight loss