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GHK-Cu supports this transition at the tissue level by modulating the gene expression patterns associated with the metabolically inflamed state that obesity research consistently identifies in adipose tissue

It activates beta-3 adrenergic receptors located in fat tissue, which triggers lipolysis (fat breakdown) without sending any signals to the brains hunger centers in the hypothalamus

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[DOI] [PubMed] [Google Scholar] 118.Matson CA, Reid DF, Ritter RC