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That celiac gene sits in the same space, and what happens with mold-susceptible clients is, all you need to, like, as far as, like, genetics go, theyre not able to mount an adaptive immune response, and further, that just allows the innate immune system to keep firing and produce histamine, and I really see that relationship with mast cell activation syndrome, even after theyve left a water-damaged building, if they have mold-susceptible haplotypes, the adaptive immune system never comes in to address the toxins in a more precise way, and so the innate immune system keeps firing, and then its like just throwing kerosene on a bonfire

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They engage overlapping but not identical pathways, serve somewhat different experimental purposes, and have very different research depth in the published literature

Respiratory Health 5.4.1.3.5