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The activation of specific signaling pathways, such as the Wnt/-catenin, PI3K/AKT, the adenosine 5-monophosphate (AMP)-activated protein kinase (AMPK), and the MAPK-ERK/1/2 pathways, has been implicated in mediating these therapeutic effects [213, 216]

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Furthermore, an increase in IGF-1 levels by around 28% was observed

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