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Here, we show that a candidate peptide, OHP2, an oral GLP-1R agonist with blood-brain barrier permeability, exhibits promising therapeutic potential for AD

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Endothelial NO synthase (eNOS) is an enzyme in endothelial cells that produces NO, a crucial signaling molecule that regulates blood vessel tone by causing relaxation (vasodilation), inhibits platelet aggregation, and prevents leukocyte (white blood cell) adhesion

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