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At the cellular level, in vitro studies have revealed that microbial metabolites directly influence kidney cell viability and inflammatory signaling

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To prevent oxidative stress and the inflammatory response in acute and chronic progressive diseases, approaches to enhance the expression of Nrf2 and thereby inhibit ferroptosis should undoubtedly play a central role in treating ALI, COPD, and idiopathic PF in the future

Nrf2 is a direct PERK substrate and effector of PERK-dependent cell survival

& Mimic-Oka, J

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