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Nonalcoholic steatohepatitis severity is defined by a failure in compensatory antioxidant capacity in the setting of mitochondrial dysfunction

doi: 10.3390/jcm11041000, 141 MenninkL

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Enhancing FAO via LCA supplementation (the cofactor of CPT1B), attenuated cardiac steatosis and lipotoxicity-induced pathological changes in the atria of obese mice, resulting in restored AF substrates and ameliorated AF susceptibility (Figure 7)

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