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This evidence establishes LA not merely as a nutrient but as a potent bioactive molecule capable of directly activating a key oncogenic pathway

Mechanistically, GLP-1RAs activated cAMP signaling in adipocytes, resulting in increased adiponectin secretion through the activation of PKA

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Interestingly, metabolomic analysis in this study indicated that semaglutide treatment upregulated L-isoleucine and methacholine or downregulated L-histidinol, arachidonic acid, and 4,5- leukotriene (LT) A4 LysoPC(16:0), which are metabolites involved in the pathogenesis of NAFLD [32]