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These alterations, secondary to heat damage, lead to increased concentrations of endotoxin and reactive oxygen species (ROS) [52], promoting significant damage to the intestinal mucosa by disrupting cellular membranes and the tight junctions [42]

To do this, they created a sort of evil FOXO4 twin called a FOXO4-DRI peptide it looked and acted like FOXO4 from a chemical perspective (in fact, p53 liked to bind with FOXO4-DRI even more than regular FOXO4, which is important because of some complicated chemistry), but FOXO4-DRI didnt only live in the nucleus it could exist anywhere in the cell

[Accessed on April 26, 2017]