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This localized depletion is strongly correlated with increased ventricular lactate, pointing to severe neuroinflammation, an overwhelmed central nervous system antioxidant network, and a direct physiological explanation for the profound cognitive dysfunction and "brain fog" experienced by patients

The VEGFR2 pathway and nitric oxide synthesis via the Akt-eNOS axis promote angiogenesis, restoring blood supply to damaged nerve tissue

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