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We hypothesized that low glutathione redox status would be associated with elevated markers of oxidative protein and DNA damage, inflammation and mitochondrial superoxide production in two regions that have been reported to be abnormal in autism, cerebellum and BA22

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We have infected normal and GSH-depleted HeLa cells with PV1-wt and PV1-BSOr and analyzed progeny virus and packaging intermediates by sucrose gradients (Figure 7)