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Once gout has been diagnosed, conventional medical treatments include: Drugs for acute attacks (non-steroidal anti-inflammatory drugs [NSAIDs], corticosteroids, colchicine) Drugs to reduce hyperuricemia (xanthine oxidase inhibitors like allopurinol to decrease production of uric acid, uricosuric drugs to increase excretion of uric acid) Emerging Therapeutics Most mammals are able to convert uric acid to allantoin with the enzyme uricase and then easily excrete it through the kidneys

Zhou and Cai [51] demonstrated significant neurological improvement in a group treated with stent placement and mechanical fat aspiration

Understanding the links between inflammatory mechanisms, tendon homeostasis and resolution of tendon damage will be crucial in developing novel therapeutics for human tendon disease

Patients with pre-existing eye disease should discuss this risk with their provider

Which patients or situations are better suited for oral versus injectable semaglutide when considering weight loss

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