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It is proposed that the non-ubiquitinated NRF2 would remain bound to the KEAP1-Cul3 complex and therefore prevent the newly translated NRF2 from inhibition by the E3-ubiquitin complex, thereby promoting NRF2 accumulation in the nucleus [17]

Patients receiving NAC had tissue reactions and tumor responses from radiotherapy judged to be similar to a control group

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Before we can even begin to talk about skin, we have to understand the compound itself

We speculated that GSH may inhibit high glucose-induced PSC activation via the ROS/TGF/SMAD pathway, thereby inhibiting fibrosis progression