PCSK9 inhibition, a posttranslational regulator of LDLR degradation, improves hepatic steatosis, inflammation, and CVD outcomes in mice and humans [43,44]
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Indeed, the combination of D + Q is more effective and allows to target more senescent cell types (compared to the use of each drug alone), by inhibiting the pro-survival pathways induced in senescent cells, notably in idiopathic pulmonary fibrosis (IPF) 10,11,12 , diabetes and chronic kidney disease 13 or Alzheimers disease 14
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